Parkinson’s disease is a complex neurological condition. Growing evidence suggests toxic exposure in the workplace and environmental pollution may play a significant role in its development. One toxic chemical drawing increased scrutiny is trichloroethylene (TCE), a common chemical compound in products ranging from industrial degreasers to paints and common household cleaners. It is also often found in contaminated soil, water and air.
Recent studies point to a potential link between long-term exposure to TCE and a heightened risk of Parkinson’s, particularly among older adults. Parkinson’s is a progressive movement disorder that worsens over time, causing pain and disability. While not directly fatal, its complications can be life-threatening, and there is currently no cure. This connection raises serious concerns for industrial workers who may have lived near or worked in factories and sites where TCE was used or released into the environment. The findings highlight how legacy pollution, often invisible and decades old, can silently affect the health of industrial workers and communities exposed to toxic chemicals like TCE.
A new report in The Lancet Neurology highlights 2023 research that found a 70% higher risk of Parkinson’s among more than 170,000 military members who served at Camp Lejeune, where the water had been contaminated with TCE, as opposed to service members at other non-contaminated locations. The report goes on to point out that the longer developmental cycle of Parkinson’s indicates we may see more of these diagnoses emerge in years to come. Considering millions of American workers were exposed to TCE at the height of its industrial use, and the widespread environmental contamination of this persistent chemical, we must consider the danger beyond just those who served at Camp Lejeune.
A different study published in 2025 in Neurology analyzed Medicare data from 2016 to 2018. Focusing on individuals aged 67 and up, the study traced residential exposure to outdoor TCE levels back in 2002 using EPA pollution maps. By comparing those diagnosed with Parkinson’s to similar individuals without the disease, and adjusting for factors like age, smoking, and air pollution, the study found patterns suggesting increased risk in areas with higher TCE emissions. Detailed mapping around major polluting sites revealed localized hotspots, reinforcing concerns about the long-term neurological impact of environmental toxins.
As research continues to uncover the hidden dangers of occupational and environmental toxins and pollutants, the emerging connection between TCE exposure and Parkinson’s disease merits increased scrutiny and investigation. This evidence adds weight to growing concerns about how long-term exposure to industrial chemicals may silently impact neurological health. Understanding these risks is a crucial step toward protecting vulnerable populations and seeking justice for those already harmed.
